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This is REQUIRED for more muscle and awesomeness
Sent Friday, February 11, 2011
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From Mike T Nelson and ExtremeHumanPerformance.com
Sent by permission only to VIP member
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Happy Friday! I woke up to -1 F temps again. Argh. I hear a
warming front is on the way and it may hit 14F today! Oh boy!
Jodie and I had a blast last night and I am working on getting a
photo up and details, but my blog is down now. Bugger.
I have the IT people looking at right now though (hopefully it will
be back up by the time you get this). Gotta love it!
My car is also in the shop again for a check engine light too. At
this rate, I hope this email gets to you!
Today's Tip: The stimulus is VERY important.
Rember we have been talking about the 3 things to reach your goals
of more muscle, strength and less fat in record time.
They are
1) Exercise 2) Nutrition 3) Accountability
There is a reason I put exercise #1 (even though ALL of them are
very important).
Without the correct stimulus, the body will adapt in the WRONG direction.
When we shoot someone into space, their body will see a MASSIVE
loss of muscle and bone.
Why?
There is not even gravity working on the body! Their body goes
"well, screw this, I don't need all this muscle and bone tissue to
get stuff done here in space!"
Years ago I took a whole class on Aerospace Physiology which is
like Anti-Exercise Physiology as I learned how everything got
smaller and weaker (atrophy)!
Gym Time
We all know that loading the muscles by weight lifting is the best
way to make them bigger and stronger.
Nothing new there.
But, did you know how powerful that stimulus really is?
What if took away ALL your anabolic hormones (testosterone and its
friends). What would happen?
What if we took out your thyroid all together?
What if you are diabetic?
.......would that stop your muscle growth?
NOPE!
Amazing! The body is so smart and redundant that even under those
conditions, with the CORRECT stimulus (exercise program) you will
STILL see progress!
Wow!
Of course we can accelerate that progress by doing many other
things correctly no doubt; but if we can get the stimulus (exercise
program) correct, you can make massive changes.
It amazes me how this information is loss in the modern age where
people (out there, not here) argue about how a 10% increase in
testosterone is the one key to the kingdom of his
hyoooooooooooogeness.
What's Old is New
If you are an uber geeks, I placed the full study at the end for
you.
You will notice it was conducted in 1975!
That was 1 year after I was born, so 35 years ago!
True, it was rat study but imagine enrolling people for a study
where day 1 it involves cutting 2 muscles in your lower leg
promptly followed by castration the next day.
I've volunteered for many crazy studies as a test subject like
getting a needle stuck in a nerve to measure signals (nerve
traffic), Wingate tests, squats done with multiple cameras and
force plates, Bod Pod, Underwater Weighing, blood flow measurements
with nitroglycerin, tilt table tests, VO2 max, HRV, and others, but
I am staying far far away from volunteering for that study.
Summary
Today's tip is to that without exercise, the odds of you reaching
your goal of more muscle, strength and less fat are pretty non
existent. No news there.
What is "new" is just how powerful exercise done CORRECTLY really
is to make massive changes in your body.
Time to lift!
Rock on
Mike N
PS
If you are confused about what program to start with, I highly
recommend the Lean Hybrid Muscle program and today is the LAST day
to pick up at 50% off as the offer expires at 12:00AM Midnight Eastern TONIGHT.
Reference
Med Sci Sports. 1975 Fall;7(3):185-98.
Mechanism of work-induced hypertrophy of skeletal muscle.
Goldberg AL, Etlinger JD, Goldspink DF, Jablecki C.
Abstract
Skeletal muscle can undergo rapid growth in response to a sudden
increase in work load. For example, the rat soleus muscle increases
in weight by 40% within six days after the tendon of the
synergistic gastrocnemius is sectioned. Such growth of the
overworked muscle involves an enlargement of muscle fibers and
occasional longitudinal splitting. Hypertrophy leads to greater
maximal tension development, although decreased contraction time
and reduced contractility have also been reported. Unlike normal
developmental growth, work-induced hypertrophy can be induced in
hypophysectomized or diabetic animals. This process thus appears
independent of growth hormone and insulin as well as testosterone
and thyroid hormones. Hypertrophy of the soleus can also be induced
in fasting animals, in which there is a generalized muscle wasting.
Thus muscular activity takes precedence over endocrine influences
on muscle size. The increase in muscle weight reflects an increase
in protein, especially sarcoplasmic protein, and results from
greater protein synthesis and reduced protein breakdown. Within
several hours after operation, the hypertrophying soleus shows more
rapid uptake of certain amino acids and synthesis of
phosphatidyl-inositol. By 8 hours, protein synthesis is enhanced.
RNA synthesis also increases, and hypertrophy can be prevented with
actinomycin D. Nuclear DNA synthesis also increases on the second
day after operation and leads to a greater DNA content. The
significance of the increased RNA and DNA synthesis is not clear,
since most of it occurs in interstitial and satellite cells. The
proliferation of the non-muscle cells seems linked to the growth of
the muscle fibers; in addition, factors causing muscle atrophy
(e.g. denervation) decrease DNA synthesis by such cells. In order
to define more precisely the early events in hypertrophy, the
effects of contractile activity were studied in rat muscles in
vitro. Electrical stimulation enhanced active transport of certain
amino acids within an hour, and the magnitude of this effect
depended on the amount of contractile activity. Stimulation or
passive stretch of the soleus or diaphragm also retarded protein
degradation. Presumably these effects of mechanical activity
contribute to the changes occuring during hypertrophy in vivo.
However, under the same conditions, or even after more prolonged
stimulation, no change in rates of protein synthesis was detected.
These findings with passive tension in vitro are particularly
interesting, since passive stretch has been reported to retard
atrophy or to induce hypertrophy of denervated muscle in vivo. It
is suggested that increased tension development (either passive or
active) is the critical event in initiating compensatory growth.
PPS
Don't procrastinate much longer as the Hybrid Muscle program for
50% off expires at 12:00AM Midnight Eastern TONIGHT. If you
want to become Hybrid athletes with strength, size, endurance,
mobility and definition just click here now before it's too late!
___________________________________________________________
Mike T Nelson
MSME, CSCS, PhD(c)
PhD Candidate, Exercise Physiology
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